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. 2017 Mar 31;49(3):e310. doi: 10.1038/emm.2016.169

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Copyright © 2017 The Author(s)

This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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An IL-20R1 deficiency reduced the severity of STZ-induced diabetic nephropathy. (a) Blood glucose measurements in IL-20R1^+/+, IL-20R1^+/− and IL-20R1^−/− mice from day 5 to 49 after STZ treatment (n=8 per group). *P<0.05 compared with IL-20R1^+/+ mice. The data are expressed as the mean±s.e.m. of three independent experiments. (b) Survival in each group (n=8 per group) was monitored daily until the end of the experiment. The data are representative of three independent experiments. (c, d) Serum BUN levels and renal glomerular areas on day 49 were analyzed in IL-20R1^+/+, IL-20R1^+/−, and IL-20R1^−/− mice after STZ or control buffer treatment (n=8 per group). *P<0.05 compared with IL-20R1^+/+ mice. The data are expressed as the mean±s.e.m. of three independent experiments.