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(a) Transferring the Hi‐expressor thyrotrophin receptor (TSHR) A‐subunit transgene from BALB/c mice to non‐obese diabetic (NOD).H2h4 mice by repeated back‐crossing. (b). Increasing percentage of NOD.H2h4 background genes from 0% in BALB/c (N0) to 98.4% in N6 offspring (adapted from data in 24).
