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. 2017 Apr 7;17:248. doi: 10.1186/s12885-017-3216-6

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — LncRNA XIST regulates HCC via miR-181a. a and b The regulation of XIST on PTEN was mediated by miR-181a. * vs Blank, p < 0.05. ** vs Blank, p < 0.01. ^## vs miR-181a mimics or miR-181a inhibitor group, p < 0.01. c and d The regulation of XIST on HCC proliferation was mediated by miR-181a. ** vs si-XIST or pcDNA-XIST group, p < 0.01. ^## vs miR-181a mimics or miR-181a inhibitor group, p < 0.01. e and f XIST inhibited the migration and invasion of HCCLM3 cells, while the transfection of miR-181a mimics abolished the inhibition. lncRNA, long non-coding RNA; HCC, hepatocellular carcinoma. ^## vs si-XIST or pcDNA-XIST group, p < 0.01