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. 2017 Apr 1;34(7):1364–1381. doi: 10.1089/neu.2016.4569

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© Fengshan Yu, et al., 2017; Published by Mary Ann Liebert, Inc.

This Open Access article is distributed under the terms of the Creative Commons Attribution Noncommercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits any noncommercial use, distribution, and reproduction in any medium, provided the original author(s) and the source are credited.

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FIG. 6. — r-mTBI does not alter myelination at 6 weeks post-injury. (A and B) Immunohistochemistry for myelin oligodendrocyte glycoprotein (MOG) to study myelination status post-imaging. No change of myelination was visible in either corpus callosum or cortex after r-mTBI (B) compared to r-sham (A), which was confirmed by quantification of MOG (E and G). (C and D) Oligodendrocyte progenitors, detected by immunolabeling for NG2 (C and D), also were not changed in either corpus callosum (F) or cortex (H) after r-mTBI compared to r-sham. Dashed lines outline the corpus callosum. n = 6 for MOG; n = 3 for NG2; values are mean ± standard error of the mean; scale bars = 50 μm. NG2, neural antigen 2; r-mTBI, repetitive mild traumatic brain injury; r-sham, repetitive sham.