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. 2017 Feb 23;8(2):e2627. doi: 10.1038/cddis.2017.51

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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(a) BCL10 inhibitory peptide represses NF-κB signaling upon PAMPs recognition in NHEK. NHEK were liposome-delivered with a BCL10 inhibitory peptide (i-BCL10) or a control scrambled peptide (c-BCL10) and exposed to PAMPs for 24 h. Expression level of the indicated NF-κB responsive genes was determined by real-time PCR. Data shown are representative of six independent experiments done in triplicate. Right panel: FITC-conjugated i-BCL10 peptide was used to monitor delivery efficiency (≅50%). (b) The expression levels of BCL10, MALT1 and CARMA2sh in NHEK left untreated or i-BCL10-delivered were assessed by western blot