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. 2016 Nov 23;21(5):1013–1023. doi: 10.1111/jcmm.13032

Figure 1.

Figure 1

NcRNAs regulate cardiomyocyte apoptosis after acute myocardial infarction. (A) MiRNAs mediate cardiomyocyte apoptosis. Pro‐apoptotic miRNAs are marked in blue including miR‐15, miR‐497, miR‐208a, miR‐34a, miR‐24, miR‐874 and miR‐155. Anti‐apoptotic miRNAs are marked in red including miR‐210, miR‐214, miR‐1, miR‐21, miR‐149, miR‐133a, miR‐499 and miR‐130a. (B) LncRNAs/circRNAs interact with miRNAs to modulate cardiomyocyte apoptosis. Pro‐apoptotic lncRNAs/circRNAs are marked in blue including APF, NRF and CDR1as. Anti‐apoptotic lncRNAs are marked in red including CARL and H19. APF, autophagy‐promoting factor; ATG7, autophagy‐related protein 7; ALDH2, aldehyde dehydrogenase 2; Apaf‐1, apoptotic protease‐activating factor‐1; Bcl‐2, B‐cell lymphoma 2; BAX, Bcl‐2‐associated X protein; BIM, BCL2‐like 11 apoptosis facilitator; CARL, cardiac apoptosis‐related lncRNA; CDR1as, cerebellar degeneration‐related protein 1 transcript; FADD, Fas‐associated protein with death domain; NRF, necrosis‐related factor; PI3K, phosphoinositide‐3‐kinase; PTP1B, protein tyrosine phosphatase‐1B; PTEN, phosphatase and tensin homolog; PARP, pro‐apoptotic gene poly ADP‐ribose polymerase; PHB2, prohibitin‐2; RIPK1, receptor‐interacting serine/threonine protein kinase 1; RIPK3, receptor‐interacting serine/threonine protein kinase 3; TNF‐α, tumour necrosis factor‐α.