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. 2017 Apr 12;8:217. doi: 10.3389/fphys.2017.00217

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Copyright © 2017 Roy and Finley.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Predicted response to metabolic perturbations. The model predicts the relative number of pancreatic cancer cells when the activity of the target enzyme was reduced by 85% alone or in combination with 85% GOT1 knockdown. The targets investigated were (A) GLUT1, (B) OXPHOS, (C) GLS, (D) MALPi, (E) GAPDH, and (F) GOT2.