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. 2017 Apr 13;16:78. doi: 10.1186/s12943-017-0639-2

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — STAT5A promotes HNRNPKP2 expression. a, The levels of 29 transcription factors in BGC823 cells after DC-SIGNR knockdown by real-time PCR (up). The levels of 29 transcription factors in SGC7901 cells after DC-SIGNR overexpression by real-time PCR (middle). Western blot confirmed the protein level of STAT5A in BGC823 cells after DC-SIGNR knockdown and in SGC7901 cells after DC-SIGNR overexpression (down). b-c Real-time PCR confirmed the STAT5A expression and western blot analysis of STAT5A protein level in BGC823 cells after DC-SIGNR knockdown with si-STAT5A and in SGC7901 cells after DC-SIGNR overexpression with pCMV3-STAT5A. D, HNRNPKP2 expression in BGC823 cells after DC-SIGNR with si-STAT5A and in SGC7901 cells after DC-SIGNR overexpression with pCMV3-STAT5A is analyzed by real-time PCR