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. 2017 Apr 13;16:78. doi: 10.1186/s12943-017-0639-2

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — CXCR4 is significantly disregulated after DC-SIGNR knockdown. a a heatmap of target genes (CXCL3, KMT2A, ERAP1, CXCR4, TNFRSF19 and AKAP13) after DC-SIGNR knockdown (left panel). The levels of metastasis and invasion markers in BGC823 cells after DC-SIGNR knockdown by qRT-PCR (right panel). b,the levels of metastasis and invasion markers in SGC7901 cells after DC-SIGNR overexpression by qRT-PCR. And HNRNPKP2 expression in BGC823 cells after DC-SIGNR and HNRNPKP2 knockdown was analyzed by qRT-PCR. c flow cytometry indicated CXCR4 in BGC823 cells after DC-SIGNR and HNRNPKP2 knockdown. d CXCR4 expression in BGC823 cells after DC-SIGNR and HNRNPKP2 knockdown was analyzed by qRT-PCR