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. 2017 Mar 27;114(15):3939–3944. doi: 10.1073/pnas.1612943114

Fig. 3.

Fig. 3.

GluA knockdown leads to defects in sensory-organ differentiation and metamorphosis. (A and D) Samples injected with control 5mis MO. (B and E) MO against ciGluA. (C and F) MO against ciGluA rescued with the MO-resistant ciGluA construct. (A–C) Hatched larval DIC images merged with arrestin immunostaining. The arrestin signal was almost completely absent in MO-injected larvae (bracket in B). (D–F) Juvenile images showing merged DIC and phalloidin staining (F-actin). The arrow and asterisk in D–F indicate the endostyle and digestive tract, respectively, showing that internal organs developed normally. The bracket in E indicates the tail that failed to shrink. The brackets in D and F indicate normal, shrunken tails. [Scale bars, 50 μm (A and D).] (G) Percentage of larvae that swam in indicated light conditions. Mean ± SE, **P < 0.001, *P < 0.005.