Table 2.
Sensory neurons examined for osmotic upshifts
| Mutated gene/transgene | Affected neurons | Number of turns/min for the 5th minute | p value | |||
|---|---|---|---|---|---|---|
| Wild-type control | Mutant/transgenic | |||||
| 200# mOsm | 400 mOsm | 200# mOsm | 400 mOsm | |||
| ttx-1 | AFD | 10.40 ± 0.79 (30) | 22.20 ± 2.35 (20) | 9.86 ± 0.80 (28) | 21.15 ± 1.86 (27) | 0.863 |
| che-1 | ASE | 14.36 ± 1.21 (28) | 30.31 ± 1.72 (32) | 18.19 ± 1.62 (32) | 28.28 ± 1.41(32) | 0.057 |
| Psrh-142::twk-18 | ADF | 6.75 ± 0.73 (8) | 26.00 ± 2.41 (8) | 8.57 ± 1.65 (7) | 24.25 ± 1.47 (8) | 0.298 |
Values are reported as the mean ± SEM (n animals tested). Mutants defective in the development and/or function of several sensory neurons are examined for osmotic upshifts. ttx-1 and che-1, as well as the wild-type controls for these two mutants, are tested at 200 and 400 mOsm for osmotic upshifts. The transgenic animals that express Psrh-142::twk-18(gf) and the nontransgenic siblings are tested at 150 and 400 mOsm for osmotic upshifts. The number of turns per minute is shown for the 5th minute for each genotype and osmolarity treatment. The interaction of genotype and treatment is tested with two-way ANOVA on the number of turns per minute in the 5th minute. The number of animals tested under each condition is indicated in the parenthesis next to mean ± SEM.
ttx-1 and che-1, as well as the wild-type controls for these two mutants, were tested at 200 and 400 mOsm for osmotic upshifts. The transgenic animals that express Psrh-142::twk-18(gf) and the nontransgenic siblings are tested at 150 and 400 mOsm for osmotic upshifts.