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. 2017 Jan 18;13(3):1695–1701. doi: 10.3892/ol.2017.5614

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Copyright: © Lin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Effects of LCA on the cell cycle progression of gastric cancer cells. (A) SGC-7901 and (B) MKN-45 cell lines were exposed to BL, LCA and LCA combined with 5-FU for 48 h. The cell cycle distributions of SGC7901 cells when exposed to (C) blank control, (D) LCA (which induced growth inhibition associated with G2/M arrest) and (E) LCA combined with 5-FU (which induced G₀/G₁ arrest more markedly) for 48 h are presented. All assays were performed in triplicate. BL, blank control; LCA, licochalcone A; 5-FU, 5-fluorouracil.