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. 2017 Jan 4;13(3):1183–1188. doi: 10.3892/ol.2017.5566

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Copyright: © Tang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — mRNA expression levels of WIF-1, APC, β-catenin and cyclin D1 in non-small cell lung cancer tissue samples and paracarcinoma tissue samples. The mRNA levels of WIF-1, APC, β-catenin and cyclin D1 were determined by reverse transcription-quantitative polymerase chain reaction, and β-actin served as the loading control. (A) Representative images of mRNA expression in cancerous tissues from adenocarcinoma or squamous carcinoma cases and in adjacent tissues to the carcinoma. (B) Analysis of the ratios of each target band density to that of β-actin in each case for four genes in normal tissues and cancerous tissues. Data are shown as means ± standard deviation. *P<0.05 compared with the normal group (n=52). WIF-1, Wnt inhibitory factor-1; APC, adenomatous polyposis coli; mRNA, messenger RNA.