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. 2017 Jan 20;13(3):851–860. doi: 10.3892/etm.2017.4071

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Copyright: © Zhu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — ICa density-voltage associations in control, adjacent and axotomized neurons were examined by the application of 80-msec voltage steps to various test potentials between −70 and +40 mV in 10-mV steps with 5-sec intervals from a holding potential of −90 mV. The ICa of adjacent (104±17 pA/pF; n=5) and axotomized neurons (75±17 pA/pF; n=8) was significantly lower than that of control neurons (144±24 pA/pF; n=7; P<0.05). In addition, the ICa of axotomized neurons was lower than that of adjacent neurons (P<0.05). The activation voltage of ICa in control and adjacent neurons was 0 mV, while that in axotomized neurons was −10 mV. Values are expressed as the mean ± standard deviation. *P<0.05 vs. control neurons. ^#P<0.05 regarding adjacent vs. axotomized neurons. ICa, Ca²⁺ density; pA/pF, peak current density; C, control; SNL, spinal nerve ligation; L4, adjacent neurons; L5, axotomized neurons.