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. 2017 Apr 26;11:62. doi: 10.3389/fnbeh.2017.00062

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Copyright © 2017 Han, Jiang, Ding, Liu, Xiao and Shi.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Expression of stathmin and tubulin in the hippocampus. (A) Dual-immunofluorescence image showing stathmin-ir and glial fibrillary acid protein (GFAP)-ir in the hippocampus of the control group. (B) A higher magnification image showing colocalization of stathmin-ir and GFAP-ir in the hippocampus of the control group. (C) Dual-immunofluorescence image showing stathmin-ir and NeuN-ir in the amygdala of the control group. (D) Dual-immunofluorescence image showing stathmin-ir and NeuN-ir in the cingulate cortex of the control group. (E) A higher magnification image showing colocalization of stathmin-ir and NeuN-ir in the amygdala of the control group. (F) A higher magnification image showing decreased stathmin in the amygdala of the SPS group. (G,H) Expression of tubulin in the amygdala of the control group (G) and the SPS group (H). (I,J) Colocalization of stathmin- and tubulin-ir in the hippocampal CA1 region of control (I) and SPS group (J). The magnification image of colocalzation of stahtmin- and tubulin-ir were showed in the I-a (merge), I-b (stathmin), I-c (tubulin0 and I-d (DAPI; *P < 0.05 vs. the control group; Bar in (B,D–F,I,J: 100 μm; Bar in A,C,G,H: 20 μm).