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. 2017 Mar 2;37(5):949–956. doi: 10.1161/ATVBAHA.116.308763

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© 2017 The Authors.

Arteriosclerosis, Thrombosis, and Vascular Biology is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 5. — In patients the response of reticulated platelets to ADP is inhibited to a greater extent by ticagrelor than by clopidogrel. The reticulated platelet subpopulation among nonaggregated single platelets was assessed by flow cytometry in platelet-rich plasma incubated with vehicle, ADP, or arachidonic acid (AA). Samples were obtained from patients taking (A) ticagrelor or (B) clopidogrel (in addition to aspirin). Representative flow cytometric images of ADP-stimulated platelet aggregates (platelets red; mRNA green) formed in samples from patients taking aspirin plus (C) ticagrelor or (D) clopidogrel. Scale bars, 7 μm. Data presented as individual data points with overlaid mean±SEM and compared by paired t test (n=9–10; ***P<0.001).