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Proposed mechanism(s) of acute i.v. injection of relaxin in rat mesenteric arteries. Relaxin injection causes rapid (3 h) and sustained (24 h) augmentation of BK‐evoked relaxation. The enhanced BK‐mediated relaxation is dependent on the IKCa channels 3 h after acute i.v. relaxin injection. In contrast, the sustained (24 h) relaxation to BK is dependent on PGI2.
