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. 2017 Apr 20;169(3):470–482.e13. doi: 10.1016/j.cell.2017.04.003

Figure 5.

Figure 5

CHN-1 Ligase Activity Regulates DAF-2 Degradation and Longevity

(A and B) CHN-1::FLAG expression rescues the short lifespan of chn-1(by155) mutant worms; catalytic inactive CHN-1(U-box)::FLAG does not.

(C) DAF-2 protein level increases during aging in analyzed daf-2 mutant alleles. Importantly, daf-2(gk390525) allele displays increased DAF-2 level already at day 1 of adulthood.

(D) Short lifespan of chn-1(by155) is suppressed by daf-2(e1368) loss-of-function mutant.

(E) Deletion of chn-1 increases level of DAF-2e1368 mutant protein, analyzed by immunoblotting of worm lysates of indicated age. (F) daf-2(gk390525) worms exhibit short lifespan.

(G) Depletion of DAF-2 by RNAi extends the short lifespan of daf-2(gk390525) allele.

(H) Deletion of chn-1 does not further increase the level of DAF-2K1614R mutant protein (daf-2(gk390525) allele), analyzed by immunoblotting of worm lysates of indicated age.

(I) Loss of chn-1 shortens lifespan of daf-2(gk390525) allele to the level of chn-1(by155) worms. See Table S1 for lifespan statistics.

See also Figures S4 and S6.