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. 2017 Apr 20;130(8):906–913. doi: 10.4103/0366-6999.204101

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Copyright: © 2017 Chinese Medical Journal

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as the author is credited and the new creations are licensed under the identical terms.

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Morphological changes observed by immunofluorescence and H and E staining (n=9 in each group). (a) Contrastive immunofluorescence of IgA deposition in the glomeruli of kidney sections from normal rats and rats with IgA nephropathy (×450). (b) Representative H and E staining of glomeruli in kidney sections from rat of each group (×400). (c) Glomerular cell number expressed as cells per glomerular cross-section of all groups stained by HE. *Compared to Control_TAK242, P < 0.05; ^†Compared to IgAN_TAK242, P < 0.05; ^‡Compared to Control_Pio, P < 0.05; ^§Compared to IgAN_Pio, P < 0.05. IgAN: Immunoglobulin A nephropathy; Pio: Pioglitazone; TAK242: Toll-like receptor 4 inhibitor.