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. 2017 Mar 18;7(5):645–651. doi: 10.1002/2211-5463.12199

Figure 3.

Figure 3

MiR‐29a‐3p enhanced the drug responses of laryngocarcinoma by PROM1. (A–B) Effect of miR‐29a‐3p on cell proliferation was measured by MTT assay after miRNA infection in TU212 and M2E cells and DDP treatment (10 μm). (C–D) miR‐29a‐3p promoted laryngocarcinoma cell apoptosis by PROM1. TU212 and M2E cells were transfected with miR‐29a‐3p or PROM1 lentivirus and then treated with DDP (10 μm). Cell apoptosis was analyzed by flow cytometry. Data assessed from three independent experiments and the P values were calculated by t‐test (*P < 0.05; **P < 0.01).