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. 2017 May 1;9:121. doi: 10.3389/fnagi.2017.00121

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Copyright © 2017 Zhang, Zhang, Zhan, Li, Zhou, Xing, Jiang, Chen and An.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Increment of Ace-H3K9 and Ace-H4K12 levels in HDAC1-, HDAC2- or HDAC3-silenced SH-SY5Y cells. siRNA duplex was used to interfere with endogenous HDAC1, HDAC2 or HDAC3 mRNA expression (untreated cells and non-specific siRNA as controls). The relative levels of Ace-H3K9 and Ace-H4K12 were analyzed by Western blot in HDAC1-silenced (A,B), HDAC2-silenced (C,D) or HDAC3-silenced (E,F) cells. (n = 6; mean ± SD; One-way ANOVA followed by LSD multiple comparison tests; ^**p < 0.01 vs. control groups).