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. 2014 Jul 7;28(9):1571–1584. doi: 10.1210/me.2014-1101

Table 2.

MR-Interacting Peptides, Derived From Heart- and Kidney-Specific cDNA Libraries, Identified Using T7 Phage Display

Clone Symbol Gene Name Fragment Activity on MR
HEK293
H9c2
A F A F
Heart A10 SFRS18 Splicing factor, arginine/serine-rich 18 474–696
A1b NUCKS1 Nuclear casein kinase and cyclin-dependent kinase substrate 1 53–243
A20 NOLC1 Nucleolar and coiled-body phosphoprotein 1 105–324
A38 RBBP6 Retinoblastoma binding protein 6 872–1056
A44 EIF5B Eukaryotic translation initiation factor 5B 160–294
A44b SSRP1 Structure-specific recognition protein 1 526–709
A53 NPIPL3 Same as D15 but longer fragment 665–854 n.d. n.d. n.d. n.d.
A78 ATRX α-Thalassemia/mental retardation syndrome X linked 193–327
A9 SAFB Scaffold attachment factor B 4–127
A91 AHNAK AHNAK nucleoprotein 899–1114
D1 BUB3 Budding uninhibited by benzimidazoles 3 homolog 220–328
D15 NPIPL3 Nuclear pore complex interacting protein-like 3 748–854
D17b CCDC55 Coiled-coil domain containing 55 296–451
D30 Hypothetical Similar to AGAP002076-PA 299–331
D32b ATP5I ATP synthase, H + transporting 14–66
F18b RPL4 Ribosomal protein L4 402–462
F19b MYL2 Myosin, light chain 2, regulatory, cardiac, slow 96–153
F20b XRCC6 X-ray repair complementing defective repair in CHO cells 6 480–609
F45b RRBP1 Ribosome binding protein 1 homolog 180 kDa 79–246
F46b RPL23A Ribosomal protein L23a 42–71
F5b SERF2 Small EDRK-rich factor 2 02–059
F6 EEF1A1 Eukaryotic translation elongation factor 1α1 396–462
F81 ENSA Endosulfine-α 17–121
Kidney KA14a MUC1 Mucin 1, cell surface associated 38–115
KA3b FRMD4B FERM domain containing 4B 528–643
KA44b Chloride intracellular channel 1 variant 90–133
KF 22b GPX3 Glutathione peroxidase 3 62–111
KF18a RRBP1 Ribosome binding protein 1 homolog 180 kDa 102–160
KF24b PCBP2 Poly(rC) binding protein 2 198–285
KF67b EEF1A1 Eukaryotic translation elongation factor 1α1 59–92 n.d. n.d. n.d. n.d.

Abbreviations: n.d., not determined; –, no effect; ↑, enhance MR transactivation; ↓, reduce MR transactivation.