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. 2017 May 3;14:97. doi: 10.1186/s12974-017-0870-1

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — The immunohistochemical staining of iNOS (a to c) and COX-2 (e to g) 48 h after SCI. The quantitative analysis of iNOS and COX-2 from spinal cord tissues in each experimental group has been assessed; bar graphs represent the mean optical density of iNOS (d) and COX-2 (h), respectively. iNOS and COX-2-positive apoptotic cells (i and j, respectively) in spinal cord lesions were also quantified by counting the total number of positive nuclei through entire cross sections by an independent observer. Quantitative analysis was based on at least five fields per section of five rats, separately. (*P < 0.05 vs. sham group; **P < 0.01 vs. sham group; ^▲ P < 0.05 vs. sham group; P < 0.01 vs. control group; #P < 0.01 vs. control and sham group)