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. 2017 Mar 14;7(3):e1059. doi: 10.1038/tp.2017.32

Table 3. Pathway analysis of the risk genes for BD.

Canonical pathways P-value Overlap Genes
Glucocorticoid receptor signaling 1.94E−03 4/270 (0.015) MED1, GTF2A1, HSPA1L, TAF15
Glutamine biosynthesis I 1.98E−03 1/1 (1) GLUL
Estrogen receptor signaling 2.07E−03 3/128 (0.023) MED1, GTF2A1, TAF15
β-alanine degradation I 3.96E−03 1/2 (0.5) ALDH6A1
Assembly of RNA polymerase II complex 4.42E−03 2/50 (0.04) GTF2A1, TAF15
Protein ubiquitination pathway 1.38E−02 3/254 (0.012) NADJC7, HSP1L, UBE2E1
Xenobiotic metabolism signaling 1.41E−02 3-256 (0.012) MED1, ALDH6A1, DNAJC7
Oleate biosynthesis II (animals) 1.97E−03 1/10 (0.1) ALDH6A1
Aryl hydrocarbon receptor signaling 2.94E−02 2/135 (0.015) MED1, ALDH6A1
Valine degradation I 3.51E−02 1/18 (0.056) ALDH6A1
Aldosterone signaling in epithelial cells 3.60E−02 2/151 (0.013) DNAJC7, HSPA1L
DNA methylation and transcriptional repression signaling 3.90E−02 1/20 (0.05) ARID4B
B-cell development 4.47E−02 1/23 (0.043) IL7R

Abbreviation: BD, bipolar disorder.