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. Author manuscript; available in PMC: 2018 Feb 20.
Published in final edited form as: Neuroscience. 2016 Dec 22;343:384–397. doi: 10.1016/j.neuroscience.2016.12.021

Figure 3.

Figure 3

Performance of untreated controls (Veh), hormone-withdrawn ‘postpartum’ females (PpD), simulated proestrus animals (ProE), and hormone-treated ‘postpartum’ rats (Horm) in the active escape test. Panel A provides information about the fluctuation of escape latencies during the 30-trial test, with each tracing point representing the average escape latency of five consecutive trials. Panel B demonstrates the mean escape latency of all 30 trials. One-way ANOVA found a significant hormone treatment effect (F3,48=6.66 P<0.001). Escape latencies of hormone-treated ‘postpartum’ rats were shorter relative to those of untreated controls and hormone-withdrawn ‘postpartum’ females (*P<0.01, Tukey-Kramer test) but not significantly different from those of simulated proestrus animals (P=0.196, Tukey-Kramer test). Panel C displays the number of failed trials (Escape Failures) during the same 30-trial active escape test. Kruskal-Wallis oneway ANOVA on ranks revealed a significant hormone treatment effect (H=18.665 df=3 P<0.001). Hormone-treated ‘postpartum’ rats made less escape failures than animals in other groups (#P<0.002, Mann-Whitney U test).