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. 2017 Mar 30;15(5):2983–2990. doi: 10.3892/mmr.2017.6416

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Copyright: © Zhou et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effects of ERY on the expression of MAPK phosphorylation in CS-induced emphysema in rats by western blotting. (A) The protein expression of MAPKs and p-MAPKs as measured by western blotting. (B) For p-p38, p-ERK1/2, ERK1/2, p-JNK and JNK, both bands were quantified in the densitometry. Densitometrically analyzed target p-MAPKs bands normalized to MAPKs. Data are presented as the mean ± standard deviation. Control rats were exposed to normal air and received daily saline injections. CS rats were exposed to CS for 12 weeks. CS + ERY rats were exposed to CS for 12 weeks and received daily injections of 100 mg/kg ERY. *P<0.05 vs. control group and ^ΔP<0.05 vs. CS group; n=12 per group. MAPK, mitogen-activated protein kinase; p-, phospho-; ERK, extracellular signal-regulated kinase; JNK, c-Jun N-terminal kinase; CS, cigarette smoke; ERY, erythromycin.