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. 2017 Apr 11;37(5):2652–2662. doi: 10.3892/or.2017.5573

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Copyright: © Ouyang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — GINS2 promotes cervical cancer cell proliferation and tumorigenicity. (A) Western blot analysis of GINS2 expression in GINS2-silenced SiHa and HeLa cells. GAPDH was used as the loading control. (B) Colony formation assay showing that silencing of endogenous GINS2 inhibited proliferation. (C) Anchorage-independent growth ability assay showing that GINS2 knockdown suppressed cervical cancer cell tumorigenicity. The number of colonies >0.1 mm in diameter was quantified after 10 days of culture. Original magnification, ×200. Bars represent the mean ± SD of 3 independent experiments; *P<0.05.