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. 2017 Apr 6;7:693. doi: 10.1038/s41598-017-00782-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of SAA1 on the activation of MAPKs and NF-κB in human amnion fibroblasts. (A–C) Effect of SAA1 (10 ng/mL; 15, 30, 60, 120 and 240 min) on the phosphorylation of p38 (A, n = 4), ERK1/2 (B, n = 3) and JNK (C, n = 4) in human amnion fibroblasts. (D,E) Effect of SAA1 (10 ng/mL; 15, 30, 60,120 and 240 min) on the abundance of phosphorylated p65 (D, n = 4) and total IκBα (E, n = 3) in human amnion fibroblasts. Top panels are the representative immunoblots. Data are the means ± SEM. Statistical analysis was performed with one-way ANOVA test. *P < 0.05, **P < 0.01, ***P < 0.001 vs. control (0).