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. 2017 Apr 6;7:686. doi: 10.1038/s41598-017-00790-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Role of potassium channels in SF-induced relaxation of aortic rings. Endothelium-intact rings were incubated with cumulative doses of SF in the absence (Salvia plus vehicle; filled circles) or presence of (A) 10 µM of Glibenclamide (Glib + Salvia; squares), or (B) Tetraethylammonium (100 µM) (TEA + Salvia; triangles). n = 6 or 5 for glibenclamide or TEA experiments, respectively. In both cases, no difference was noted between inhibitor treated and vehicle administered curves; p > 0.05 for Salvia alone versus either Glib + Salvia or Salvia + TEA.