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. 2017 Apr 7;7:755. doi: 10.1038/s41598-017-00933-y

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(a–d) Histograms showing frequency of HMOX-genotypes (classified as SS, SM, SL, MM, ML) stated in the BiliHealth study population. Figure 2 shows statistical distribution and frequency of respective HMOX-genotypes, expressed as (GT)_n allelic mean, between the study groups. (a and b) Male and female (all) subjects (Phenotype: GS: dark grey; C: light grey). (c and d) Female subjects only (Phenotype: GS: dark grey; C: light grey). Data distribution proposes a non-significant higher abundance of SM alleles in GS individuals, whereas the MM variant appeared to be more prominent in C subjects. This non-significant observation suggests a (gender-specific) higher frequency of shorter GT-repeats in GS individuals, which would be worth pursuing in a larger study collective. Classification of GT-fragment lengths (S < 26, L > 30) was performed following Chang et al.⁷².