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. 2017 Apr 20;7:981. doi: 10.1038/s41598-017-01056-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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TTX and eleclazine (Elec) abolished ventricular arrhythmias induced by Bay K 8644 both in absence (a) and presence (b) of ATX-II. Representative recordings of MAPs (upper records in each panel) and ECG (lower records in each panel) were recorded simultaneously in control, Bay K 8644 (200 and 300 nM, a) or ATX-II-treated hearts (b) before and after treatment with TTX. Arrows indicate an episode of VT. The incidence of all ventricular arrhythmias and VT are presented in panel c. ^* P < 0.05 vs. ctrl; ^# P < 0.05 vs. Bay K 8644; ^† P < 0.05 vs. ATX-II; ^& P < 0.05 vs. ATX-II plus Bay K 8644.