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. 2017 Apr 21;7:1007. doi: 10.1038/s41598-017-01249-7

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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DAC reduces the expression of surface markers of DCs from VKH patients. Immature DCs from VKH patients were cultured with or without the presence of DAC for 6 days, then followed by stimulation with 100 ng/mL LPS for 24 hours. DCs were subsequently harvested for flow cytometry analysis with specific surface antibodies against CD80, HLA-DR, CD86, CD83 and CD40. (A) Histograms with overlays are from representative experiments of DAC treated and untreated DCs from VKH patients. (B) Mean fluorescence intensity (MFI) of surface markers on DAC treated and untreated DCs from VKH patients. Data are shown as mean ± SEM. Statistical analysis was performed using the paired-samples t-test (n = 8, *p < 0.05; **p < 0.01).