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. 2017 Mar 14;13(5):2873–2880. doi: 10.3892/ol.2017.5862

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Glucose metabolism (glycolysis and aerobic oxidation) and the hypoglycemic mechanism of biguanides. AMP, adenosine monophosphate; ATP, adenosine triphosphate; FBP1, fructose-bisphosphatase 1; GLUT-2, solute carrier family 2 member 2; G6Pase, glucose-6-phosphatase; GCK, glucokinase; GYS, glycogen synthase 1; PYGL, phosphorylase, glycogen, liver; PFKL, phosphofructokinase, Liver Type; PEPCK, phosphoenolpyruvate carboxykinase 2, mitochondrial. Reprinted by permission from Macmillan Publishers Ltd.: Nature Reviews Endocrinology (9), copyright (2014).