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. 2017 May 16;45(6):e600–e606. doi: 10.1097/CCM.0000000000002310

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Copyright © 2017 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the Society of Critical Care Medicine and Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 1. — Endoscopy and histology in antibiotic-associated apoptotic (AAA) enterocolitis. (A) Hemorrhagic inflammation with loss of folds in the duodenum (top). Complete loss of vascularization, “geographic” ulcerations with abundant fibrin and denudation in the colon (bottom). (B) Segmental loss of crypts and villi and abundant apoptoses (arrows) in the duodenal mucosa (inset: activated caspase-3 positivity). (C) Large areas completely devoid of crypts and surface epithelium in the colon (inset: crypt epithelial apoptosis). Activated caspase-3 positive crypt epithelial cells and cell debris (bottom). Images: A (top) and B, case A onset of diarrhea; A (bottom), case C 45 d after diarrhea onset; C (top), case C 15 d after diarrhea onset; C (bottom) and inset C, case C 17 d after onset of diarrhea.