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. 2017 May 16;6:48. doi: 10.1186/s13756-017-0206-z

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Minimum spanning tree of VRE isolates. Minimum spanning tree of 71 VRE patients (P, yellow) and environmental (E, green) isolates from haematology/oncology unit and one reference isolate (non-oncology ward, isolated in May 2015) based on 1423 cgMLST target genes, pairwise ignoring missing values. Genotypes are consecutively numbered, starting with P1 (isolated in March 2015). Each dot represents one genotype. Size of dots correlates with the number of identical genotypes. Connecting lines show the number of alleles differing between two genotypes (thick line: ≤20 alleles, thin line 21–40 alleles and dotted line >40 alleles). Whole Genome Sequencing revealed five clusters of VRE, two vanA-clusters and three vanB-clusters