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. 2017 Apr 12;15(6):3699–3705. doi: 10.3892/mmr.2017.6466

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Copyright: © Yang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — H/R modulates the expression level of miR-15a in H9c2 cells. Reverse transcription-quantitative polymerase chain reaction analysis indicated that miR-15a expression in H9c2 cells was upregulated in the H/R group, when compared with the control (normoxia) group. Results were normalized to U6 and expressed as the fold change relative to the control. Data are presented as the mean ± standard deviation from three independent experiments. ^{^^}P<0.01 vs. normoxia. H/R, hypoxia/reoxygenation; miR-15a, microRNA-15a.