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The effects of c-Met inhibitor foretinib on cell viability, survival, phosphorylation of c-Met and downstream PI3K/Akt pathway in ESCC cells. (A) Foretinib inhibited cell proliferation in a time and dose-dependent manner in ESCC cells. (B) The clonogenic survival of ESCC cells was suppressed significantly by foretinib at 0, 2, 4, 6, 8 Gy. (C) Western blotting results demonstrated that foretinib blocked the phosphorylation of c-Met and Akt obviously.
