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. 2017 May 19;10:251. doi: 10.1186/s13071-017-2183-8

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — PvGAMA antigenicity during natural malaria infection. The dot plot shows OD distribution (Y-axis) for detecting rPvGAMA-Nt or rPvGAMA-Ct by P. vivax (a) or rPvGAMA-CT by P. falciparum (b) infected (I) and non-infected (NI) patients’ sera (X-axis). rPvGAMA-Nt: infected individuals n = 37, m ± SD = 0.38 ± 0.29; control individuals n = 6, m ± SD = 0.12 ± 0.1. rPvGAMA-Ct: infected individuals n = 37, m ± SD = 0.67 ± 0.32; control individuals n = 6, m ± SD = 0.14 ± 0.08. rPvGAMA-Ct recognised by P. falciparum infected patients’ sera: infected individuals n = 30, m ± SD = 0.06 ± 0.04; control individuals n = 10, m ± SD = 0.06 ± 0.03