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. 2017 Jan 24;9(2):91–103. doi: 10.1093/jmcb/mjx008

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© The Author (2017). Published by Oxford University Press on behalf of Journal of Molecular Cell Biology, IBCB, SIBS, CAS.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Involvement of the ECS–NO^• signalling axis in disease development and/or treatment. Schematic outlines the reported involvement of ECS–NO^• signalling in the tissues/processes presented. The participation of different ECS components (i.e. CB1R, CB2R, or TRPV1) is inferred from the use of selective agonists and/or through genetic or pharmacological inhibition, and is presented in brackets if known. Also included are details of the specific NOS isoform(s) involved in mediating the indicated response if determined. Arrows denote an inferred induction (↑) or repression/alleviation (↓) of the indicated process/pathology in response to activation of the specified ECS component.