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. 2017 May 22;112(4):39. doi: 10.1007/s00395-017-0629-y

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 2 — More profound LV dysfunction in Hmox1^−/− than in Hmox1^+/+ mice after MI. At indicated time-points after MI or sham surgery a LV EF, b LV FS, c LV Vs, d LV Vd, e LV IDs, and f LV IDd were determined with TTE; n = 7–10 mice/group. g, h Evaluation of cardiomyocyte hypertrophy. g Representative fluorescent images of WGA (red) and DAPI (blue) in the peri-infarct (border zone) and remote myocardium (remote zone) of MI-operated Hmox1^+/+ and Hmox1^−/− mice. h Graph summarizing quantitative analysis of cardiomyocyte cross-sectional area (CSA) in the border and remote myocardium.*p < 0.05, **p < 0.01, ***p < 0.0001, ^†vs. appropriate sham control, ^#vs. other MI. Scale bar 50 μm