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. 2017 May 16;8:15205. doi: 10.1038/ncomms15205

Table 2. Incidence of malignant pleural effusions caused by parental and KRAS -modulated tumour cell lines.

n=206 MPE
P value
  no yes %  
LLC shC (G12C) 3 11 79 c
LLC shKras 10 2 17 0.0048
MC38 shC (G13R) 0 11 100 C
MC38 shKras 10 4 29 0.0005
AE17 shC (G12C) 1 10 91 c
AE17 shKras 11 0 0 <0.0001
FULA shC (G12V, Q61R/H) 1 8 89 c
FULA shKras 8 1 11 0.0034
B16F10 pC (WT) 10 1 9 c
B16F10 pΔKras2A 0 10 100 <0.0001
B16F10 pΔKras2B 1 12 92 <0.0001
PANO2 pC (WT) 14 2 13 c
PANO2 pΔKras2A 0 12 100 <0.0001
PANO2 pΔKras2B 4 12 75 0.0010
SKMEL2 pC (WT) 9 0 0 c
SKMEL2 pΔKras2B 2 8 80 0.0007
HEK293T pC (WT) 9 0 0 c
HEK293T pΔKras2B 2 7 78 0.0023

AE17, C57BL/6 malignant pleural mesothelioma; B16F10, C57BL/6 malignant skin melanoma; FULA, FVB urethane-induced lung adenocarcinoma; HEK293T, human embryonic kidney cells; LLC, C57BL/6 Lewis lung carcinoma; MC38, C57BL/6 colon adenocarcinoma; PANO2, C57BL/6 pancreatic adenocarcinoma; SKMEL2, human malignant skin melanoma; WT, wild-type.

Shown is number of mice (n) that developed dry pleural carcinomatosis (no MPE;<100 μl pleural fluid) and number (n) and percentage (%) of mice that developed MPE (≥100 μl pleural fluid). P<0.0001 for overall comparison by χ2 test. P, probability values for comparison with parental control cells (c) by Fischer's exact tests.