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. 2017 Mar 2;13(5):1702–1710. doi: 10.3892/etm.2017.4171

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Effects of miR-98 mimics and inhibitor on the proliferation of ox-LDL-treated HUVECs. (A) The miR-98 mimics significantly promoted the proliferation of ox-LDL-treated HUVECs, whereas the miR-98 inhibitor exerted the opposite effect, as determined by cell counting kit-8 assays. (B and C) The miR-98 mimics significantly promoted the proliferation of ox-LDL-treated HUVECs, whereas the miR-98 inhibitor exerted the opposite effect, as determined by EdU assays. The second group of “vehicle” were treated with ox-LDL while the first group of “vehicle” not. *P<0.05 vs. the vehicle and ox-LDL treatment group. miR-98, microRNA-98; ox-LDL, oxidized low-density lipoprotein; HUVECs, human umbilical vein endothelial cells; oligo, oligonucleotide.