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. 2017 Mar 28;13(5):2293–2303. doi: 10.3892/etm.2017.4270

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Copyright: © Zhou et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 7. — Histomorphology of infarcted hearts. (A) Representative histological Masson's trichrome staining of transverse sections of infarcted hearts 28 days following UTMD-mediated Ang-1 gene delivery. The infarct length and thickness (blue) of the left ventricular wall are presented (no magnification). The (B) infarct length and (C) thickness of the left ventricular wall were measured 4 weeks after UTMD-mediated Ang-1 gene delivery. The TCMB and CMB groups (n=20 per group) had similar infarct lengths, which were significantly lower than that of the control group. However, the thickness of the infarct region was significantly increased in the TCMB group compared with that of the CMB and control group (n=10 in the control group). Ang-1, angiopoietin 1; UTMB, ultrasound-targeted microbubble destruction; CMB, non-targeted cationic microbubble; TCMB, targeted cationic microbubble; NS, not significant.