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. 2017 May 24;7:2348. doi: 10.1038/s41598-017-02572-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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D1R expression in the dlBST. (A) Schematic summary of major known connections of the dlBST. BLA, basolateral nucleus of the amygdala; CeA, central nucleus of the amygdala; INS Cx, insular cortex; PVT, paraventricular nucleus of the thalamus; SNc, substantia nigra pars compacta ^{24, 43, 44}. (B) Timeline of experimental manipulations. D, day. (C) Representative dlBST mapping of D1R expression (dashed lines) in sham-operated (sham), 6-OHDA-lesioned (Non-Dysk) and L-Dopa-treated dyskinetic 6-OHDA-lesioned rats (Dysk) (scale bar: 300 µm) with an inset showing a magnification of D1R expression in dyskinetic condition (scale bar: 5 µm) (STR = Striatum). (D) Representative insets (scale bar: 20 µm) showing D1R, ∆FosB and co-localization of D1R/∆FosB expression.