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Ineffectiveness of AR antagonists to reverse dormancy established after a transient exposure to R1881. (A and C) Schemes of the experimental design for AR antagonist treatments. (B and D) Cloning efficiencies of the cells cultured as indicated. Values are the mean ± sd of 2 independent experiments. Only GSK (8 mM GSH, 0.2 µM SB50512 and 0.2 µM K02288) treatment displayed a significant effect on the reversal of androgen-induced dormancy (black asterisks).
