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. 2017 May 25;12(5):e0177900. doi: 10.1371/journal.pone.0177900

Fig 2. TORC1 signaling regulates migration to low salt concentrations through S6K.

Fig 2

(A) A schematic diagram of putative TORC1 signaling in C. elegans. (B) Salt chemotaxis of wild-type N2 worms and rsks-1(ok1255) mutants after salt conditioning. (C) Expression of rsks-1 cDNA under the pan-neuronal H20 promoter, but not ASER-specific gcy-5 promoter or intestinal ges-1 promoter, rescued the chemotaxis abnormality of rsks-1(ok1255) mutants. Salt chemotaxis was tested after high-salt/food(–) conditioning. (D) Salt chemotaxis of wild-type N2 animals and atg-13(bp417) mutants after salt conditioning. *p < 0.05, **p < 0.01, n.s. = not significant (one-way ANOVA with Dunnett’s post hoc test, N ≥ 9).