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. 2017 May;33(3):241–249. doi: 10.6515/ACS20160902A

Table 2. Clinical and angiographic characteristics between patients with or without AAC.

All patients (n = 225) AAC (+) (n = 127) AAC (–) (n = 98) p-value
ACS type
 STEMI 78 (35) 40 (32) 38 (39) 0.26
 NSTEMI 132 (59) 80 (63) 52 (53) 0.13
 UA 15 (7) 7 (5) 8 (8) 0.43
TIMI scores 4.2 ± 2.3 4.9 ± 2.4 3.3 ± 1.7 < 0.001
Peak
 CK (U/L) 1094 ± 1883 986 ± 1996 1235 ± 1727 0.33
 CK-MB (U/L) 70.9 ± 88.7 62.6 ± 80.2 81.9 ± 98.1 0.12
 Troponin-I (ng/ml) 34.9 ± 100.1 24.4 ± 45.3 48.9 ± 143.2 0.12
CAD
 SVD 42 (19) 19 (15) 23 (24) 0.03
 DVD 54 (24) 33 (26) 21 (22) 0.96
 TVD 115 (51) 66 (52) 49 (50) 0.5
 Insignificant 9 (4) 8 (6) 1 (1) 0.11
 Normal CAG 5 (2) 2 (1) 2 (2) 0.42
LM disease 27 (12) 20 (16) 6 (7) 0.04
LVEF 44 ± 16 43 ± 17 45 ± 15 0.66
Killip class 2.1 ± 1.1 2.4 ± 1.1 1.7 ± 1.0 < 0.001
Revascularization 190 (84) 105 (83) 85 (87) 0.46

Data are represented as mean ± standard deviation or n (%).

AAC, aortic arch calcification; CAD, coronary artery disease; CAG, coronary artery angiography; CK, creatine kinase; DVD, double vessel disease; LM, left main; LVEF, left ventricular ejection fraction; NSTEMI, non-ST elevation myocardial infarction; STEMI, ST elevation myocardial infarction; SVD, single vessel disease; TIMI, thrombolysis in myocardial infarction; TVD, triple vessel disease; UA, unstable angina.