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. 2017 Feb 1;7(1):175–185. doi: 10.1086/690206

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© 2017 by Pulmonary Vascular Research Institute

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 4. — (a, b) HighK⁺-induced contraction is not different between PAs from CH and DH mice; however, EDR is significantly attenuated in DH-PAs compared to CH-PAs. (a) HighK⁺-induced pulmonary arterial contraction. n = 5 per group. Data are mean ± SEM. (b) ACh-induced pulmonary arterial relaxation. CH, n = 7; DH, n = 6. Data are mean ± SEM. *P < 0.05 vs. CH. (c, d) Pretreatment of mitoT enhanced EDR in DH-PAs, but not in CH-PAs. (c) ACh-induced pulmonary arterial relaxation in the presence and absence of mitoT in CH. n = 4 per group. Data are mean ± SEM. (d) ACh-induced pulmonary arterial relaxation in the presence and absence of mitoT in DH. n = 4 per group. Data are mean ± SEM. *P < 0.05 vs. CH.