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. 2017 May 31;8:904. doi: 10.3389/fpls.2017.00904

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Copyright © 2017 Martinelli and Dandekar.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The three main molecular mechanisms driving symptom progression of Huanglongbing disease in citrus: (A) source-sink disruption due to starch accumulation in leaves, stimulated by upregulation of glucose-phosphate-transport2 (GPT) and the induction of genes involved in photosynthetic reactions in fruits; (B) non-beneficial modulation of jasmonic acid-salicylic crosstalk that is not correlated with the different pathogen concentration in leaf and fruit tissues; and (C) modulated expression of detoxifying proteins linked with different susceptibility of citrus genotypes. Red color means “upregulated,” green means “downregulated” while white means “not-regulated.” Genes not mentioned in the text were: NBS-LRR (nucleotide binding site leucine rich repeats), lox2, and lox3 (lipoxygenase2 and 3), GTS (glutathione-S-transferases).