The lack of myogenic tone in mouse bladder feed arterioles is not due to endothelial NO production, increased TRPV4 channel activity, or KCa channels. A: representative tracings of bladder feed arteriole responses to 300 nM apamin + 100 nM charybdotoxin (ChTx) (black trace), 100 µM l-NNA (blue trace), or 10 nM GSK2193874 (red trace). B: inhibition of KCa channels (apamin/ChTx), eNOS (l-NNA), or TRPV4 channels (GSK2193874) had no effect on bladder feed arteriole diameter. N = 4–9.