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. 2017 Feb 1;312(5):F836–F847. doi: 10.1152/ajprenal.00682.2016

Fig. 3.

Fig. 3.

The lack of myogenic tone in mouse bladder feed arterioles is not due to endothelial NO production, increased TRPV4 channel activity, or KCa channels. A: representative tracings of bladder feed arteriole responses to 300 nM apamin + 100 nM charybdotoxin (ChTx) (black trace), 100 µM l-NNA (blue trace), or 10 nM GSK2193874 (red trace). B: inhibition of KCa channels (apamin/ChTx), eNOS (l-NNA), or TRPV4 channels (GSK2193874) had no effect on bladder feed arteriole diameter. N = 4–9.